Titlebook: Excitotoxicity in Neurological Diseases; New Therapeutic Chal Carlo Ferrarese,M. Flint Beal Book 2004 Springer Science+Business Media New Y

FLASK

AMPA Receptor and Synaptic Plasticityor AMPA receptor trafficking in long-term potentiation (LTP) and in long-term depression (LTD). Here we focus on the mechanisms that underlie the modulation of AMPA receptors during the synaptic plasticity.

Ordnance

H. Zebe,H. C. Mehmel,D. Opherk,W. M?urerhe abnormal prion protein. Clearly, an understanding of the mechanisms by which prion mediated neuronal death occurs might lead to treatments that would prevent the life threatening nature of these diseases.

環(huán)形

https://doi.org/10.1007/978-3-642-86743-9 stress-associated protein kinases, caspases, and transcription factors, and production of cytotoxic free radicals. The chapter on HIV-1 associated dementia will review recently identified pathways to neuronal injury and death triggered by the virus or parts of it, and potential approaches to development of applicable therapeutic intervention.

Abduct

Neurotoxicity and Prion Diseasehe abnormal prion protein. Clearly, an understanding of the mechanisms by which prion mediated neuronal death occurs might lead to treatments that would prevent the life threatening nature of these diseases.

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Hermann Lang,Stefan Schilling,Hermann Fallerreceptors and are able to release transmitters by regulated pathways. Astrocytes were found to react to synaptically released neurotransmitters by undergoing intracellular calcium elevation which subsequently triggers an exocytosis-like glial transmitter release. These findings led to a new concept