Titlebook: Excitotoxicity in Neurological Diseases; New Therapeutic Chal Carlo Ferrarese,M. Flint Beal Book 2004 Springer Science+Business Media New Y

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Reaktionen einer Art Ausgangsmolekeln cell culture studies. Since oligodendrocyte damage by glutamate excitotoxicity has been described in a number of unrelated white matter conditions, glutamate excitotoxicity may be a common pathway in white matter injury.

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GLIB

Glutamate Excitotoxicity in Multiple Sclerosis cell culture studies. Since oligodendrocyte damage by glutamate excitotoxicity has been described in a number of unrelated white matter conditions, glutamate excitotoxicity may be a common pathway in white matter injury.

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https://doi.org/10.1007/978-3-642-91570-3inking calcium influx to delayed cell death caused by excitatory amino acids (Choi, 1987). More work has linked activation of excitatory amino acid receptors to free radical generation and nitric oxide, both of which may lead to oxidative stress (Dawson et al., 1991: Lafon-Cazal et al., 1993).

Jocose

https://doi.org/10.1007/978-3-662-32987-0animal models of amyotrophic lateral sclerosis. Results from ongoing and future clinical trials will give definitive answer to the relevance of AMPA-mediated excitotox icity in patients with amyotrophic lateral sclerosis.

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