Titlebook: Cellular Ca2+ Regulation; Douglas R. Pfeiffer,Jeanie B. McMillin,Steve Littl Book 1988 The Editor(s) (if applicable) and The Author(s), un

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Psychologische Probleme in Organisationen,servations are related to the mechanism(s) through which the mitochondrion contributes to Ca. homeostasis in the normal cell remains to be determined. However, under pathological conditions such as ischemia this process of Ca. release may relate to matrix Ca. content and mitochondrial volume changes observed (4).

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Die Organisation als komplexes System, synthesis (Beg et al., 1985), and alterations in cytoskeletal organization and function (Ben-Ze’ev, 1985). Given these and other observations of alterations of cellular processes associated with malignancy, the general hypothesis has surfaced that tumor cell calcium metabolism is abnormal.

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Purification of Ca2+-ATPase from Rat Pancreatic Acinar Plasma Membranes Using Calmodulin-Affinity Chgher levels of calcium (approximately 10. M) in the extracellular space (Peterson, 1982). Calcium therefore penetrates into cells continuously down its electrochemical gradient by passive diffusion. In order to maintain a homeostatic environment in the cell and assure proper cell function, free calcium must be continuously removed.

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Book 1988rnational Congress of Physiological Sciences which occurred in Vancouver, canada, at that time. 2 Adjustments in the cytoplasmic Ca + concentration of cells occur in response to a variety of external signals. These fluctuations are a cen- tral component of one mechanism by which cells adapt their ac

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