Titlebook: Cellular Ca2+ Regulation; Douglas R. Pfeiffer,Jeanie B. McMillin,Steve Littl Book 1988 The Editor(s) (if applicable) and The Author(s), un

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James G. March,Herbert A. Simonc reticular membranes. Each of these membranes possesses distinctive Ca. transport processes by means of which it regulates cytosolic Ca. concentration.. While Ca. mobilization into the cytosolic compartment subsequent to cell activation plays an important role in the elicitation of various normal c

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Die Organisation als komplexes System,attern of metabolism. Many of these alterations involve processes modulated by the intracellular level and distribution of calcium. In addition to increased growth rates, Ca. may play a specific role in the increased rates of aerobic glycolysis (Cittadini et al., 1981), elevated rates of cholesterol

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https://doi.org/10.1007/978-3-322-93840-4s attracted much interest (Trump and Berezesky, 1984; Schanne et al., 1979; Jewell et al., 1982; Lowrey et al., 1981; Bellomo and Orrenius, 1985). Clearly, many cellular processes are regulated or influenced by the cytosolic free calcium concentration, which in turn is controlled by calcium transpor

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https://doi.org/10.1007/978-3-322-93840-4 in the endoplasmic reticulum. Fig. 1 shows the various ATP-dependent pumps and antiporters involved in the control of Ca.. and Na.. (In this figure, the plasmalemmal Na.-Ca. antiporter is conventionally assumed to operate in the forward mode i.e. Ca. efflux energized by Na. influx). Since Ca. trans

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https://doi.org/10.1007/978-3-322-93857-2f such agonists are norepinephrine and epinephrine acting via α.-adrenergic receptors, vasopressin acting on V. receptors, ATP and ADP acting on P. purinergic receptors and angiotensin II. Each agonist, after binding to its specific cell surface receptor, provokes the hydrolysis of PI 4,5-P. by a ph

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https://doi.org/10.1007/978-1-4757-0007-7cell; cell membrane; cells; cytoplasm; development; distribution; environment; ion transport; iron; membrane;