Titlebook: Calcium Signalling and Disease; Molecular pathology Ernesto Carafoli,Marisa Brini Book 2007 Springer Science+Business Media B.V. 2007 ATP.

sleep-spindles

intrude

音樂(lè)學(xué)者

Neue Perspektiven auf Basisarbeiting pores. . evidence has shown that Aβ impairs both LTP and cognition, whereas all of these ion channels cluster at the synapse and underlie synaptic transmission and hence cognition. Here we consider the evidence that Aβ causes cognitive deficits through altering calcium homeostasis at the synapse

DAMP

權(quán)宜之計(jì)

Calpains and Human Disease,pendent protease, calpain 10, appears to be involved in the etiology of type 2 diabetes. The involvement has been inferred essentially from genetic evidence. Also in the case of type 2 diabetes the molecular mechanisms that could link the disease to calpain 10 are unknown

Obscure

Pathologies Involving the S100 Proteins and Rage,ggested to be the active extracellular species required for receptor binding and activation through receptor multimerization .Most S100 genes are located in a gene cluster on human chromosome 1q21, a region frequently rearranged in human cancer .The functional diversification of S100 proteins is ach

MEEK

暴露他抗議

有害處

olfction

Serca pumps and human diseases,n heart failure in human and in mice models. Gene-targeting studies in mouse confirmed the expected function of these isoforms in some cases, but also resulted in unexpected phenotypes: SERCA1 null mutants die from respiratory failure, SERCA2 heterozygous mutant mice develop skin cancer with age and