Titlebook: Calcium Signalling and Disease; Molecular pathology Ernesto Carafoli,Marisa Brini Book 2007 Springer Science+Business Media B.V. 2007 ATP.

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https://doi.org/10.1007/978-3-642-58996-6e conditions linked to dysfunctions of cellular Ca . homeostasis. The evidence supporting their involvement has traditionally been indirect and circumstantial, but recent work has added more solid evidence supporting the role of ubiquitous dimeric calpains in the process of neurodegeneration. The on

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Karl-Heinz Erdmann,Johanna Frommbergerel of Ca._o and to respond with changes in function that are directed at normalizing the blood calcium concentration. In addition to the parathyroid gland, the kidney is a key site for Ca._o-sensing that enables it to make physiologically relevant alterations in divalent cation and water metabolism.

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https://doi.org/10.1007/978-3-658-27206-7r contributions of voltage-gated calcium channels in multiple physiological processes. Moreover, we are beginning to comprehend their pathophysiological roles through both naturally occurring channelopathies in humans and mice and through targeted gene deletions. The data illustrate that small pertu

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Neues Denken in der Unternehmensführungdiseases. This chapter discusses these diseases in terms of known mechanisms, controversies, and unanswered questions. We also compare the cardiac and skeletal muscle diseases to explore common mechanisms

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