Titlebook: Bone Toxicology; Susan Y. Smith,Aurore Varela,Rana Samadfam Book 2017 Springer International Publishing AG 2017 Bone Pharmacology.Bone Phy

CULP

Hectic

Allodynia

Bone and Muscleen the factors regulating both bone and muscle, including factors like myogenic regulatory factors (MRFs) and myostatin from muscle, bone-derived factors like bone morphogenetic proteins (BMPs) and prostaglandin E. (PGE.), as well as other secretory factors, are discussed to provide a better underst

填料

Pituitary Hormone-Driven Mechanism for Skeletal Lossand and/or receptor yields a skeletal phenotype with the primary target organ remaining unaffected. Recognition and in-depth analysis of the mechanism of action of each pituitary hormone has improved our understanding of bone pathophysiology and opens new avenues for therapy. Here we discuss the int

優(yōu)雅

Kidney–Bone: Interactionlites, as well as blood electrolyte status and (2) bone-derived factors including FGF23, MEPE, and osteocalcin. Dysfunction in any of these factors alone or in combination can disrupt a multitude of the mineral balance in the cross-organ communication between the kidneys, intestine, bones, and parat

Sigmoidoscopy

Bone and the Immune System formation. Specifically, the bone anabolic actions of parathyroid hormone and the pharmaceutical costimulation inhibitor CTLA4-Ig (abatacept) are mediated though mechanisms involving T-cells. This chapter documents some of the key discoveries in the field of osteoimmunology in the context of both p

ARCHE

Bone and Energy Metabolismdulate adipose tissue function and pancreatic islet cells production of insulin in the pancreas. The central nervous system is the final common pathway modulating these signals, both in the skeleton and in adipose tissue. Disruption in this complex regulatory circuit and its downstream tissues is ma

Pillory

Projection

2168-4219 eans to develop the concepts in part three which describes bone and its interaction with other organ systems.? The unique series of chapters in part three, contributed to by key leaders in their respective fiel978-3-319-85848-7978-3-319-56192-9Series ISSN 2168-4219 Series E-ISSN 2168-4235