Titlebook: Urothelial Carcinoma; Methods and Protocol Michèle J. Hoffmann,Nadine T. Gaisa,Thorsten H. Ec Book 2023Latest edition The Editor(s) (if app

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書目名稱Urothelial Carcinoma影響因子(影響力)




書目名稱Urothelial Carcinoma影響因子(影響力)學(xué)科排名




書目名稱Urothelial Carcinoma網(wǎng)絡(luò)公開度




書目名稱Urothelial Carcinoma網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Urothelial Carcinoma被引頻次




書目名稱Urothelial Carcinoma被引頻次學(xué)科排名




書目名稱Urothelial Carcinoma年度引用




書目名稱Urothelial Carcinoma年度引用學(xué)科排名




書目名稱Urothelial Carcinoma讀者反饋




書目名稱Urothelial Carcinoma讀者反饋學(xué)科排名

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A Drug Repurposing Pipeline Based on Bladder Cancer Integrated Proteotranscriptomics Signaturesisease heterogeneity and the limitations of the current therapeutic modalities, such as drug low efficacy and patient resistance acquisition. Drug repurposing is a cost-effective strategy that targets the reuse of existing drugs for new therapeutic purposes. Such a strategy could open new avenues to

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glomeruli

Using Sister Chromatid Exchange Assay to Detect Homologous Recombination Deficiency in Epigeneticalld chromatids and their sisters can be visualized in cells when DNA synthesis in one chromatid is labelled by 5-bromo-2’-deoxyuridine (BrdU). Homologous recombination (HR) is considered as the principal mechanism responsible for the sister chromatid exchange (SCE) upon replication fork collapse, and

d-limonene

Identification of STAG2-Mutant Bladder Cancers by Immunohistochemistryand are therefore classified as non-muscle-invasive bladder cancer (NMIBC). A minority of tumors are diagnosed after they have invaded the underlying detrusor muscle and are classified as muscle-invasive bladder cancer (MIBC). Mutational inactivation of the STAG2 tumor suppressor gene is common in b

Sarcoma

Genome-Wide CRISPR Screening for the Identification of Therapy Resistance-Associated Genes in Urotheide screening, which has some advantages over other screening methods using chemical DNA mutagens for inducing genetic changes, RNA interference, or arrayed screens. Here we describe the use of genome-wide knockout and transcriptional activation screening enabling the CRISPR-Cas9 system to discover

municipality

Tissue Slice Culture and Analysis of Tumor-Associated Hyaluronan in Urothelial Carcinomaerized by deregulated hyaluronan metabolism. It is postulated that the deregulated metabolism in cancer tissue is characterized by elevated hyaluronan synthesis and degradation. This results in the accumulation of small hyaluronan fragments in the tumor microenvironment which promotes cancer-related