Titlebook: Osteoporosis in Older Persons; Pathophysiology and Gustavo Duque,Douglas P. Kiel Book 2009 Springer-Verlag London 2009 Fractures.Osteoporo

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Sex Steroids and Aging Bone,ere gender-specific: estrogen was important for women and testosterone for men. However, research over the past decade has demonstrated a key role for estrogen in maintaining skeletal integrity in men. Thus, a unitary model for involutional osteoporosis has been proposed (1) that identifies estrogen

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Animal Models for Senile Osteoporosis,ast and nematode to mouse and non-human primates, and has rapidly progressed because of the recent development of forward and reverse genetics, as well as functional genomics. A number of mouse models bearing artificially or naturally modified genes develop bone phenotypes with various pathologies.

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Genetics of Osteoporosis in Older Age,rounds for the pathophysiology of invo-lutional osteoporosis: a rapid bone loss after menopause as a result of estrogen withdrawal, and a gradual age-related bone loss thereafter. Women experience a more rapid phase of bone loss after the menopause, caused mainly by estrogen deficiency ., but a less

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Fracture Epidemiology Among Individuals 75+,esidual lifetime risk of fracture has been estimated to be 44–65% for women and 25–42% for men (1). After age 75, hip fractures are the most frequent fractures, and up to 50% of older individuals suffering a hip fracture will have permanent functional disability, 15–25% will require long-term nursin

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Non-Pharmacological Treatments for Falls and Fractures,domized controlled trials (RCT) include exercise, occupational therapy interventions incorporating education and home hazard modification, psychotropic medication withdrawal, expedited cataract extraction, cardiac pacing for carotid sinus hypersensitivity, and targeted multi-factorial interventions.

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