Titlebook: New Trends in Hepatology; The Proceedings of t Paolo Gentilini,Mario Umberto Dianzani Conference proceedings 1996 Kluwer Academic Publisher

LIMN

sacrum

Lamellar Bodies in Human Gallbladder Bileae are real structures existing in bile or methodological artifacts and, b) the effect that ursodeoxycholic acid treatment exerts on biliary lipid carriers. Lamellae are real lipid structures occurring in bile as they can be isolated and characterized with different techniques, including quasielasti

外形

Hepatocyte Lipid-Metabolic Interplays and the Cholesterol Secretion Process to Bile: Insights from Rhospholipid in close association with bile acids for reasons of solubilization and transport (1,2). While bile acids are still considered the main drivers of bile lipid secretion, increasing interest has been addressed to the multiple hepatic metabolic functions in which cholesterol is involved, suc

飛來(lái)飛去真休

lian Ministry of the University, Scientific Research and Technology, having been chosen from several other scientific programs. The group is now called the National Group for the Study of Liver Cirrhosis and Viral Hepatitis and includes more than 40 centers throughout the nation under the auspices o

maudtin

4-Hydroxy-Alkenals, Products of Lipid Peroxidation, as Signals for Biochemical Functions of Normal Csue damage. However, aldehydic concentrations below 10. M, i.e. those detectable under basal conditions, exert positive effects on cell migration, activation and function. A physiological role of defined classes of biogenic aldehydes is thus proposed.

oncologist

Oxidative Damage in Chronic and Acute Liver Disease.on, DNA adducts formation in relation to free radical-mediated damage, and on glutathione metabolism, as a measure of antioxidant defense. The results presented demonstrate that oxidative damage is an important pathologic mediator in liver disease.

錯(cuò)誤

Possible Role of Ethanol-Derived Free Radicals in the Pathogenesis of Alcohol-Induced Liver Damagesociated to alcohol abuse [1]. In recent years, however, an increasing number of studies have suggested that, beside acetaldehyde, free radical intermediates might be involved in the pathogenesis of alcohol toxicity.

tenuous

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phlegm

The Genomic Response of Liver Cells to Post-Ischemic Reperfusion and Oxidative Stress.ar to the ischemic-reperfused liver: under the latter circumstance oxidative damage caused by reperfusion is clearly superimposed on molecular injuries caused by ischemia, which are mainly responsible for the activation of the heat shock factor and the induction of the major heat shock gene hsp 70.