Titlebook: New Technologies for Glutamate Interaction; Neurons and Glia Maria Kukley Book 2024 The Editor(s) (if applicable) and The Author(s), under

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Belgin Yal??n,Michelle Monjechtlich interessieren bzw. mit denen sie sich in gewisserma?en identifizieren. Es ist nun Aufgabe des Projektes Ma?nahmen zu kreieren, die sich auf diese Themen beziehen und somit in kleinen Schritten den Studenten an das Thema ?Krankenkasse“ und ?Krankenversicherung“ heranführen soll. Es werden The

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Ting-Jiun Chen,Bartosz Kula,Maria Kukleychtlich interessieren bzw. mit denen sie sich in gewisserma?en identifizieren. Es ist nun Aufgabe des Projektes Ma?nahmen zu kreieren, die sich auf diese Themen beziehen und somit in kleinen Schritten den Studenten an das Thema ?Krankenkasse“ und ?Krankenversicherung“ heranführen soll. Es werden The

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Visualization of Glutamatergic Neurotransmission in Diverse Model Organisms with Genetically Encodedynapses are dense in the vertebrate brain, at ~1/μm.. Glutamate is released from and onto diverse components of the nervous system, including neurons, glia, and other cells. Methods for glutamate detection are critically important for understanding the function of synapses and neural circuits in nor

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Surface Glutamate Receptor Nanoscale Organization with Super-Resolution Microscopy (dSTORM) in particular single-molecule localization microscopy techniques, revealed a complex organization of surface glutamate receptors, especially within the synapse. The use of direct stochastic optical reconstruction microscopy (dSTORM) provided the necessary resolution to dissect the precise localizat

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Ligand-Directed Chemical Labeling for Visualizing and Analyzing AMPA Receptors in Neuronsion of AMPA-type glutamate receptor (AMPAR) trafficking is essential for synaptic plasticity, which is a putative cellular mechanism for memory. Conventional biochemical and molecular biological approaches have been widely used for analyzing the trafficking of AMPARs. However, it is still challengin

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Live FRET-FLIM Imaging to Study Metabotropic Signaling via the NMDA Receptorat the NMDAR can induce synaptic depression without ion flux, suggesting that it has a metabotropic function. Our results show that glutamate binding or elevated amounts of beta-amyloid can trigger a conformational change in the NMDAR c-terminal domain. We have shown previously that this movement af