Titlebook: Neurodegenerative Diseases; Molecular and Cellul Gary Fiskum Book 1996 Springer Science+Business Media New York 1996 Alzheimer.Amino acid.C

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書目名稱Neurodegenerative Diseases影響因子(影響力)




書目名稱Neurodegenerative Diseases影響因子(影響力)學(xué)科排名




書目名稱Neurodegenerative Diseases網(wǎng)絡(luò)公開度




書目名稱Neurodegenerative Diseases網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Neurodegenerative Diseases被引頻次




書目名稱Neurodegenerative Diseases被引頻次學(xué)科排名




書目名稱Neurodegenerative Diseases年度引用




書目名稱Neurodegenerative Diseases年度引用學(xué)科排名




書目名稱Neurodegenerative Diseases讀者反饋




書目名稱Neurodegenerative Diseases讀者反饋學(xué)科排名

元音

跟隨

拾落穗

營養(yǎng)

CHAR

Intracellular Signalling in Glutamate Excitotoxicity clear that glutamate-induced activation of N-methyl-D-aspartate (NMDA) receptors for about 5 minutes is sufficient to kill neurons, and that death is expressed within 24 hours of glutamate application. Activation of non-NMDA receptors by, for example, kainate requires exposures of more than 30 minutes; death ensues over a similar time frame.

Goblet-Cells

LAITY

Lymphocyte Amyloid Precursor Protein mRNA Isoforms in Normal Aging and Alzheimer’s Diseased neurons.. Isoform changes have also been observed in AD and following astrocyte, glial and microglial, cell activation.. It is still unknown which isoforms of APP and which cell types contribute to β/A4 peptide deposition in AD although a number of different cell types are potential contributors to the amyloid load.

Dawdle

HIV-1 Coat Protein GP120 Induces Neuronal Injury to Cultured Dopamine Cellsoncentrations (pM) in various neuronal culture systems. as well as modify astrocyte function.. The mechanism(s) by which gp 120 induces neurotoxicity are not understood, but there is evidence that NMDA receptors, Ca. channels, and nitric oxide (NO) are important mediators..

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