Titlebook: Membrane Defenses Against Attack by Complement and Perforins; Charles J. Parker (Associate Professor of Medicine Book 1992 The Editor(s) (

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B. P. Morganlibration studies with market data.Scenario simulation frameThis book considers the one-factor copula model for credit portfolios that are used for pricing synthetic CDO structures as well as for risk management and measurement applications involving the generation of scenarios for the complete univ

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W. F. Rosse rates or respectively the cross section of bond prices. The new Heath, Jarrow and Morton (HJM) models perfectly fit to an observed initial term structure by focussing on the arbitrage-free pricing of related derivatives. Given a specification of the volatility for the forward rates or bond prices t

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E. R. Podackfresne and Goldstein [19] derived a series expansion for the pricing of swaptions assuming a 3-factor Gaussian- and CIR interest rate model. The main drawback of the above applications of the EE comes from the dependency of the series expansion on the underlying model dynamics, meaning that the Tayl

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Regulation of Complement by Membrane Proteins: An Overview,cesses of inflammation (M.-E. 1988). For example, C3a and C5a are potent anaphylatoxins, C5a is the primary mediator of neutrophil Chemotaxis, and activation (C3b) and degradation (iC3b) fragments of C3 are critically important opsonins that are recognized by specific receptors on phagocytic cells (

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Decay Accelerating Factor (CD55),ate cleavage fragments and condensation products with specific and potent biologic activity. Activated complement proteins can function as anaphylatoxins, adherence factors, chemotaxins, opsonins, and a transmembrane pore, which can be lytic (reviewed in F. and F. 1987). Regulated complement activat

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Complement Receptor Type 1,nction as a membrane inhibitor of complement activation. The CR1 present on phagocytic cells and lymphocytes has similar functions in regulation of complement activation and serves as a receptor responsible for triggering cellular activation events such as phagocytosis or Ig synthesis. These latter

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Membrane Inhibitor of Reactive Lysis,nt-mediated lysis, at least in part by restricting the formation of the membrane attack complex (MAC). MIRL is expressed by all peripheral blood hematopoietic cells, by endothelial cells, and by some cellular elements of the peripheral and central nervous systems. The erythrocytes of paroxysmal noct