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Titlebook: Membrane Defenses Against Attack by Complement and Perforins; Charles J. Parker (Associate Professor of Medicine Book 1992 The Editor(s) (

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書目名稱Membrane Defenses Against Attack by Complement and Perforins
編輯Charles J. Parker (Associate Professor of Medicine
視頻videohttp://file.papertrans.cn/631/630358/630358.mp4
叢書名稱Current Topics in Microbiology and Immunology
圖書封面Titlebook: Membrane Defenses Against Attack by Complement and Perforins;  Charles J. Parker (Associate Professor of Medicine Book 1992 The Editor(s) (
描述In this volume a comprehensive review of membrane proteinsthat regulatecomplement and perforin-mediated cytolysis ispresented. A detailed analysisis provided of thebiochemical, molecular, and functional features of theseproteins; the glycosyl phosphatidylinositol linkedproteins are covered, with emphasis placed on the functionalimportance of this type ofmembrane anchor. Further chaptersinclude reviews of the effects of complement activation oncells that are metabolically active, and of theregulationof cell killing by cytotoxic lymphocytes, consideringthesimilarities and disparities between the complement andperforinsystems. The therapeutic use of recombinant CR1for immune-mediated pathophysiological processes isdiscussed in a separate section.
出版日期Book 1992
關(guān)鍵詞Komplementsystem; Zellmembran; activation; anemia; cell; cell membrane; h?molytische; lymphocytes; membrane;
版次1
doihttps://doi.org/10.1007/978-3-642-77014-2
isbn_softcover978-3-642-77016-6
isbn_ebook978-3-642-77014-2Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
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0070-217X etween the complement andperforinsystems. The therapeutic use of recombinant CR1for immune-mediated pathophysiological processes isdiscussed in a separate section.978-3-642-77016-6978-3-642-77014-2Series ISSN 0070-217X Series E-ISSN 2196-9965
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Decay Accelerating Factor (CD55), phase and within the membranes of host cells. The structure, function, and expression of one important membrane regulator of complement, namely, the decay accelerating factor (DAF), will be reviewed here.
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