Titlebook: Lung Inflammation in Health and Disease, Volume I; Yong-Xiao Wang Book 2021 The Editor(s) (if applicable) and The Author(s), under exclusi

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Innate Lymphoid Cells in Airway Inflammation,elevation of IL-4, IL-5 and IL-13 and other inflammatory mediators, leading to increased eosinophilic inflammation. The growing evidence of ILC2 contribution in the induction and maintenance of allergic inflammation suggests that targeting upstream mediators may affect both the innate and adaptive i

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Sex-Steroid Signaling in Lung Diseases and Inflammation,ry lung diseases and to understand the mechanistic role of sex-steroids signaling in regulating these diseases. Exploring the roles of sex-steroid signaling in the regulation of lung diseases and inflammation is crucial for the development of novel and effective therapy. Overall, we will illustrate

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Interactive Roles of CaMKII/Ryanodine Receptor Signaling and Inflammation in Lung Diseases,vival, cell cycle control, and inflammation, it is possible that the relationship between ROS and CaMKII/RyRs signal complex will be necessary for understanding and treating lung diseases. Here, we review roles of CaMKII/RyRs in lung diseases to understand with how CaMKII/RyRs may act as a transduct

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Inflammation in Pulmonary Arterial Hypertension,d or skewed immune response favoring a proinflammatory environment that can lead to the infiltration of cells such as lymphocytes, macrophages, and neutrophils. Current treatment strategies focus on the dilation of partially occluded vessels; however, such techniques have not resulted in an effectiv

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