Titlebook: Iron in Central Nervous System Disorders; Peter Riederer,M. B. H. Youdim Conference proceedings 1993 Springer-Verlag/Wien 1993 Central Ner

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,The treatment of iron overload — psychiatric implications,assaemia major has dramatically improved the prognosis of patients affected. The outcome of long-term treatment, however, heavily relies on patient compliance, which is a particular problem with a chelating agent that optimally has to be given as a s.c. infusion over several hours a day. Thus the av

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Iron and neurotoxin intoxication: comparative in vitro and in vivo studies,njection of iron. The lipid peroxidation as measured by ascorbate-induced malondialdehyde (MDA) production was increased by incubation with iron, whereas the iron chelator deferoxamine inhibited the formation of MDA.

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Cytokine induced synthesis of nitric oxide from L-arginine: a cytotoxic mechanism that targets intrde is paramagnetic (has an unpaired electron) it can readily react with dioxygen and the superoxide anion to yield toxic products. As a result, the balance between beneficial and deleterious redox reactions involving nitric oxide must be tightly regulated by currently unidentified mechanisms.

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Conference proceedings 1993n discussed on several occasions. Yet little attention has been paid to iron, the most abundant transitional metal in the body and the earth‘s crust. Iron plays a major role as a cofactor of numerous metabolic enzymes, it is important for DNA and protein synthesis, and has a crucial role in the oxyg

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Some reflections on iron dependent free radical damage in the central nervous system, some difficulties scientists have to face when interpreting experimental data by which they want to prove or to rule out a role of reactive oxygen species (ROS) in the pathogenesis or progression of human diseases.