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Titlebook: Iron in Central Nervous System Disorders; Peter Riederer,M. B. H. Youdim Conference proceedings 1993 Springer-Verlag/Wien 1993 Central Ner

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21#
發(fā)表于 2025-3-25 04:57:38 | 只看該作者
Brain iron and schizophrenia,and control subjects without neuropsychiatric diseases. Analysis of iron content showed a clear regional difference with highest iron levels in the caudate nucleus. There was no correlation between iron content and the neuroleptic-free period prior to death. Iron content in the schizophrenic group w
22#
發(fā)表于 2025-3-25 08:37:30 | 只看該作者
23#
發(fā)表于 2025-3-25 15:22:10 | 只看該作者
24#
發(fā)表于 2025-3-25 19:54:26 | 只看該作者
25#
發(fā)表于 2025-3-25 20:22:13 | 只看該作者
26#
發(fā)表于 2025-3-26 00:49:36 | 只看該作者
,Intranigral iron infusion in rats: A progressive model for excess nigral iron levels in Parkinson’sc may be a direct consequence of increased iron because of iron’s ability to catalyze cytotoxic free radical formation. We have developed an animal model based on excess nigral iron in PD through intranigral infusion of iron citrate in rats. These infusions cause a long-term and dose-related increas
27#
發(fā)表于 2025-3-26 04:34:10 | 只看該作者
28#
發(fā)表于 2025-3-26 10:40:29 | 只看該作者
29#
發(fā)表于 2025-3-26 13:42:32 | 只看該作者
Consequences of intrastriatally administrated FeCl3 and 6-OHDA without and after transient cerebralontralateral turning and rotations after apomorphine administration. Treatment with alpha-tocopherol prevents the hypersensitive reaction. The sensitivity of the cerebral tissue to apomorphine following 6-OHDA treatment is enhanced when 7.5 μg FeCl. is coadministered with 6-OHDA. Only when the combi
30#
發(fā)表于 2025-3-26 19:11:58 | 只看該作者
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