Titlebook: Intestinal Tumorigenesis; Mechanisms of Develo Vincent W. Yang,Agnieszka B. Bialkowska Book 2015 Springer International Publishing Switzerl

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書目名稱Intestinal Tumorigenesis影響因子(影響力)




書目名稱Intestinal Tumorigenesis影響因子(影響力)學(xué)科排名




書目名稱Intestinal Tumorigenesis網(wǎng)絡(luò)公開度




書目名稱Intestinal Tumorigenesis網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Intestinal Tumorigenesis被引頻次




書目名稱Intestinal Tumorigenesis被引頻次學(xué)科排名




書目名稱Intestinal Tumorigenesis年度引用




書目名稱Intestinal Tumorigenesis年度引用學(xué)科排名




書目名稱Intestinal Tumorigenesis讀者反饋




書目名稱Intestinal Tumorigenesis讀者反饋學(xué)科排名

fatty-acids

Microsatellite Instability and Intestinal Tumorigenesis,s crucial to maintaining genetic integrity. When mismatch repair is defective, microsatellite instability occurs, resulting in deregulated tissue growth and tumorigenesis. Although defunct mismatch repair proteins can occur sporadically, these defects are the hallmark of Lynch syndrome, a hereditary

Insufficient

Biology of Intestinal Epithelial Stem Cells,vide a selective barrier to the noxious lumenal contents, and provide feedback to regulate digestive functions. To achieve these roles, the epithelium is composed of several distinct cell types with different capabilities, including absorptive enterocytes/colonocytes, microfold, goblet, enteroendocr

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Chronic

Inflammation and Colorectal Cancer,n, progression and metastasis of colon cancer. Colitis associated colon cancer (CAC) is a classic example of multifactorial, multi-step colorectal cancer associated with inflammatory bowel diseases. In recent years, the generation of animal models of CAC and recognition of the importance of the gut

Foam-Cells

Sphingolipids in Intestinal Inflammation and Tumorigenesis,only as structural lipids in cell, but mediate significant biologic functions as well. Sphingolipid enzymes function in the intestinal lumen to degrade exogenous dietary sphingolipids and in the enterocytes themselves to generate endogenous sphingolipids. Among the most studied bioactive sphingolipi

Cultivate

Aging: An Etiological Factor in The Development of Intestinal Tumorigenesis, the age-related rise in sporadic CRC, including altered carcinogen metabolism and the cumulative effects of long-term exposure to cancer-causing agents. In this review article we have discussed the current evidence that implicates aging as an etiological factor in the initiation and progression of