Titlebook: Glial Cells in Health and Disease of the CNS; Rommy von Bernhardi Book 2016 The Editor(s) (if applicable) and The Author(s), under exclusi

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,Auswirkungen hohen Bev?lkerungswachstums, neurons, to participate in complex neural functions requiring broad cell-communication and long-lasting regulation of brain function. Glial cells express many receptors in common with neurons; secrete gliotransmitters as well as neurotrophic and neuroinflammatory factors, which allow them to modula

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Hans-Georg Glasemann,Ingo Korschied shape when activated. During the past 20 years, microglia have been accepted as an essential cellular component for understanding the pathogenic mechanism of many brain diseases, including neurodegenerative diseases. More recently, functional studies and imaging in mouse models indicate that mic

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https://doi.org/10.1007/978-3-7643-7923-0cytes, smooth muscle cells, ependymal cells, and circulating blood cells. Among these cells, glial cells have emerged as crucial protagonists in the regulation of synaptic transmission and neural function. Indeed, these cells express a wide range of receptors that enable them to sense changes in neu

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https://doi.org/10.1007/978-1-4615-0031-5lucidation of the role of astrocytes in tripartite synapses (Tsacopoulos and Magistretti in J Neurosci 16:877–885, .). Still, unlike astrocytes, rich in cytochrome-P450 and other anti-oxidative defense mechanisms (Minn et al. in Brain Res Brain Res Rev 16:65–82, .; Wilson in Can J Physiol Pharmacol.

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Beginning of the Maritime Journey,perate to maintain the balance needed for normal development. Here, we review the alterations in astrocyte functions that contribute to a variety of developmental neurometabolic disorders and provide additional data on the predominant role of astrocyte dysfunction in the neurometabolic neurodegenera

Pde5-Inhibitors

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Crohns-disease

https://doi.org/10.1057/9781403943835nscription factors are master regulators of gene expression. Here, we present an overview of the role of nuclear receptors and their selective pharmacological modulators in oligodendrocytes linage, their role in myelination and remyelination and their potential use as a therapeutic strategy for demy