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Titlebook: Genetics and Alzheimer’s Disease; Colloque Medecine et Pierre Marie Sinet,Yvon Lamour,Yves Christen Conference proceedings 1988 Springer-Ve

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書(shū)目名稱Genetics and Alzheimer’s Disease
副標(biāo)題Colloque Medecine et
編輯Pierre Marie Sinet,Yvon Lamour,Yves Christen
視頻videohttp://file.papertrans.cn/383/382685/382685.mp4
叢書(shū)名稱Research and Perspectives in Alzheimer‘s Disease
圖書(shū)封面Titlebook: Genetics and Alzheimer’s Disease; Colloque Medecine et Pierre Marie Sinet,Yvon Lamour,Yves Christen Conference proceedings 1988 Springer-Ve
描述This volume contains the proceedings of the meeting Genetics and Alzheimer‘s Disease held in Paris, on March 25,1988, by the Fondation Ipsen pour la Recherche Therapeuti- que. This meeting was the second of the series of Colloques Medecine et Recherche devoted to Alzheimer‘s disease. The first one was held in Angers (France) on September 14, 1987; the proceedings of this meeting have already been published and are entitled Immunology and Alzheimer‘s Disease (1988, edited by A Pouplard-Barthelaix, J Emile, and Y Christen). The third Colloque Medecine et Recherche, organized in Montpellier on Sep- tember 19, 1988, dealt with neuronal grafting; the proceedings of this last meeting will be published in early 1989 (Neuronal Grafting and Alzheimer‘s Disease: Future Perspectives, edited by F Gage, A Privat and Y Christen). In each case, the Fondation Ipsen deliberately focuses attention on the most up-to-date themes, and sometimes the most controversial ones, from medical and scientific research. The genetic aspects of Alzheimer‘s Disease (AD) stands at the very forefront of research carried out in the last 2 years. It complements research using the formal approach, which has become possi
出版日期Conference proceedings 1988
關(guān)鍵詞Alzheimer; alzheimer‘s disease; attention; genetics; immunology
版次1
doihttps://doi.org/10.1007/978-3-642-73647-6
isbn_softcover978-3-642-73649-0
isbn_ebook978-3-642-73647-6Series ISSN 0945-6066 Series E-ISSN 2196-310X
issn_series 0945-6066
copyrightSpringer-Verlag Berlin Heidelberg 1988
The information of publication is updating

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,Heterogeneity in Alzheimer’s Disease: an Exercise in the Resolution of a Phenotype,st heterogeneity in Alzheimer’s disease. Since some of these features run in Alzheimer’s disease families there could be genetic heterogeneity. Understanding the mechanisms which determine the age of onset and related features will be as important as the discovery of the genes, because delay of the
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Familial Aggregation of Dementia of Alzheimer Type: Analysis from an Epidemiological Point of View,se latter studies is to examine whether an increased familial risk does exist. Case-control studies have yet to provide clear evidence for familial aggregation of DAT. The inconclusiveness of these results is due to the very small number of studies with positive results and to methodological problem
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,Molecular Genetic Strategies in Familial Alzheimer’s Disease: Theoretical and Practical Considerati Alzheimer’s disease (AD), at least in the four large pedigrees examined. Further studies have indicated that the β-amyloid gene and the superoxide dismutase-1 gene are not the site of the familial AD (FAD) mutation, and that duplication of large regions of chromosome 21 is not the pathogenetic mech
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