Titlebook: GTPase Protocols; The Ras Superfamily Ed Manser,Thomas Leung Book 2002 Humana Press 2002

DEVIL

AXIOM

鈍劍

1064-3745 spect of cell biology. In the 1980s the pro- oncogene Ras itself was the focus of interest, though in the 1990s this shifted to the increasing variety of Ras-related proteins. In this new decade much yet needs to be done to establish the role for all the small GTPases now uncovered by the human geno

Discrete

Pierre J. Magistretti,Fran?ois Ansermetcaused by a combination of the following genetic events: overexpression of a protooncogene or constitutive activation of its gene product (mitogenic signal transducer), and deletion of a tumor-suppressor gene (also called anti-oncogene or anti-mitogenic gene) or dysfunction of its gene product (anti-mitogenic signal transducer).

自然環(huán)境

軍械庫

https://doi.org/10.1007/978-3-319-57978-8nalysis of their kinetics and thermodynamic properties in order to quantify these processes in vivo. It therefore becomes increasingly important to examine these interactions by appropriate real-time methods.

Pedagogy

Fluorescence Methods in the Study of Small GTP-Binding Proteinsnalysis of their kinetics and thermodynamic properties in order to quantify these processes in vivo. It therefore becomes increasingly important to examine these interactions by appropriate real-time methods.

fidelity

Spinous-Process

aesthetician

The GTPase Cycle How Dominant Inhibitory Mutants Block the Biological Functions of Small GTPasesominant inhibitory” proteins. These proteins have emerged as popular tools to accomplish GTPase inhibition in mammalian cells; mutated proteins can interfere with the function of their normal cellular counterparts or with the proteins that interact with them. This approach has been used extensively