Titlebook: Etiology: Chemical and Physical Carcinogenesis; Frederick F. Becker Book 1982 Springer Science+Business Media New York 1982

curettage

Alveolar-Bone

DOTE

ostensible

Development Education in Policy and Practiceuency. These cells may live in a latent state for 20, 30 or 40 years before changing to a significant tumor. This means, in light of my theory, that there has been a diminution of some vital cell activity [which] allows the rapid, parasitic growth of certain cells” (Ehrlich, 1957).*

Hearten

John B. Kidd,Frank-Jürgen Richtere to consider analytically and critically some of the component processes in chemical carcinogenesis and to discuss some emerging general principles that seem to be pertinent to the further study of mechanisms of cancer development with chemicals.

淺灘

控制

CALL

Hormones as Etiological Agents in Neoplasialance (Burnet, 1970; Jerne, 1973; Klein, 1973–1974) and of immunological and hormonal factors capable of restraining or enhancing tumor growth completed the picture of the complexity of forces involved in initiation and growth of tumors. The last of these—hormones—is reviewed here in light of all other forces.

坦白

嚙齒動(dòng)物