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Titlebook: Epigenetics - A Different Way of Looking at Genetics; Walter Doerfler,Petra B?hm Book 2016 Springer International Publishing Switzerland 2

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書目名稱Epigenetics - A Different Way of Looking at Genetics
編輯Walter Doerfler,Petra B?hm
視頻videohttp://file.papertrans.cn/314/313227/313227.mp4
概述Covers aspects from epigenetic mechanisms to development and biology of diseases.With a special focus on viruses and immunology.Discusses peptides and proteins as epigenetic modulators.Includes supple
叢書名稱Epigenetics and Human Health
圖書封面Titlebook: Epigenetics - A Different Way of Looking at Genetics;  Walter Doerfler,Petra B?hm Book 2016 Springer International Publishing Switzerland 2
描述This book presents epigenetics research as a new way of looking at genetics. Topics range from basic epigenetics mechanisms to development of cells and organisms and the?biological basis of diseases. In addition the book focuses on immunology and the role of viruses in epigenetics. The last part of the book highlights proteins and peptides as epigenetic modulators.
出版日期Book 2016
關(guān)鍵詞BRD4; EBV-host cell interactions; FMR gene; lymphocyte development; oxidative stress; short biologically
版次1
doihttps://doi.org/10.1007/978-3-319-27186-6
isbn_softcover978-3-319-80087-5
isbn_ebook978-3-319-27186-6Series ISSN 2191-2262 Series E-ISSN 2191-2270
issn_series 2191-2262
copyrightSpringer International Publishing Switzerland 2016
The information of publication is updating

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Epigenetics - A Different Way of Looking at Genetics978-3-319-27186-6Series ISSN 2191-2262 Series E-ISSN 2191-2270
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Risikofaktoren der Demenz vom Alzheimer-Typ,ation to hostile and/or unpredictable environments. Lineage formation is often under epigenetic control, thus avoiding the burden of mutation. Epigenetic lineages can be formed whenever a cell-to-cell difference (e.g., generated by intrinsic noise) is propagated by a heritable feedback loop. In othe
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https://doi.org/10.1007/978-3-0348-7842-5atory lineages. In the early days, immunologists defined their functionality by expression of a small handful of signature genes. With the present extraordinary capabilities to gain genome-wide information on gene transcription, chromatin state, and transcription factor recruitment, we now come to a
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