Titlebook: Epicardial Adipose Tissue; From Cell to Clinic Gianluca Iacobellis Book 2020 Springer Nature Switzerland AG 2020 Cardiovascular risk and ep

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https://doi.org/10.1007/978-981-15-3481-2lites needed by the myocardium. Transcriptomic and proteomic studies show that it is a local source of adipokines with paracrine influence on the myocardium due to the intimate microcirculation shared by both tissues. These analyses also show that it has a role in the immune and endocrine systems af

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Margaret Burchett,Rachid Mousine,Jane Tarranws an even more distinct link with disease manifestation, supporting the hypothesis of a local impact of visceral adipose tissue on atherosclerosis development in the underlying vasculature. This book chapter gives an overview over the existing literature, described methods for quantification of EAT

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V. N. Chukanov,B. A. Korobitsinrediction of first coronary events. EAT volume is greater in subjects with incident coronary heart disease. The incidence of fatal or nonfatal coronary event significantly increased with higher EAT and remains significant even after adjustment for coronary calcium calcification score and obesity. EA

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https://doi.org/10.1007/978-3-030-27783-3inflammation, oxidative stress, atrial remodelling, and genetic factors. EAT has been shown in multiple studies to be a risk factor for development of AF and predictor of recurrence after catheter ablation. The mechanisms directly linking EAT to the pathogenesis of AF also are uncertain. Multiple ph

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Budgetary Constraints on Doctrinal Concepts,rdial fat was actually lower in subjects with systolic heart failure. Epicardial fat may incur in fibrotic changes during chronic and advanced cardiac failure. Whether EAT plays a role in the long-term prognosis of heart failure requires future investigation.

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Air Power in the Nuclear Age, 1945–82ent studies have focused on exploring the autonomic regulation of EAT as it pertains to heart disease pathogenesis and progression. In this chapter, we provide an overview of these studies, immediately following brief introductions on the regulation of global cardiac function by the two branches of

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https://doi.org/10.1007/978-3-0348-7970-5adrenal gland—paracrine secretion of adipokines from the adipose tissue surrounding the adrenal neoplasia being hormonally active. Considering the EAT characteristics of visceral adipose tissue depot, as well as periadrenal fat, correlation has been found between EAT and cardiovascular changes in ad

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Anatomy of the Epicardial Adipose Tissue,raction and crosstalk between the EAT and the myocardium. Under pathological circumstances, epicardial adipocytes display an intrinsic pro-inflammatory and atherogenic profile. A dense inflammatory infiltrate, mainly represented by macrophages, is commonly detected in epicardial fat of subjects with