Titlebook: Electrical Diseases of the Heart; Genetics, Mechanisms Ihor Gussak (Deputy Therapeutic Area Head, Profess Book 20081st edition Springer-Ver

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Caveolae and Arrhythmogenesispace. However, unlike the historical model of the “fluid mosaic” for the plasma membrane, in which integral membrane proteins are evenly distributed and free to diffuse, current knowledge suggests a more heterogeneous view of the plasma membrane in which proteins and lipids are clustered within spec

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Senescence and Arrhythmogenesisage..–. The increased susceptibility to both ventricular and atrial arrhythmias in the senescent heart occurs despite the absence of apparent disease and is exaggerated in the presence of underlying comorbidities..,. Cardiac dysrhythmias not only adversely affect the quality of life but also contrib

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Comparisons of Substrates Responsible for Atrial Versus Ventricular FibrillationC) has been challenged by recent ideas suggesting that in some cases a single high-frequency rotor (Figure 16-1B). or even a rapidly firing focus (Figure 16-1A). can underlie fibrillation. In the latter two cases, although the primary source may be firing regularly, the inability of tissues to follo

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Ihor Gussak (Deputy Therapeutic Area Head, ProfessProvides a perspective on primary (heritable) electrical disease of the heart and sudden cardiac death in cardiology, neurology, nephrology, endocrinology by emphasizing the clinical and basic aspects

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Verfügungsrecht über die Bodensch?tzey in lethal cardiac arrhythmia, and equally frustrated by the paucity of basic understanding of cardiac electricity upon which better treatment could be based. What a remarkable change has occurred since that time; there have been extraordinary advances in both the science and clinical care. I will