Titlebook: Dietary Phenylalanine and Brain Function; Richard J. Wurtman,Eva Ritter-Walker Book 1988 Birkh?user Boston 1988 brain.metabolism.perceptio

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Normungsarbeit und Zertifizierungncentrations of the same amino acids. The increase in brain concentrations of LNAA could obviously not be explained by the competition among the LNAA in plasma for the carrier-mediated transport into the brain. These findings indicate that the transport of LNAA from plasma into the brain is regulated by a β-adrenergic mechanism.

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Effect of Aspartame on Plasma Phenylalanine Concentration in Humanshese clinical studies have examined the plasma phenylalanine response when APM was given as a bolus dose in unsweetened beverage, orange juice, a sucrose solution, capsules, or a slurry or as part of a meal. The complexity of the meal feedings ranged from soup and a beverage to a hamburger and a mil

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Aspartame Consumption in Normal Individuals and Carriers for Phenylketonuria test followed by chronic intake for a period of 12 weeks. Two doses of aspartame were used in the loading test, 50 mg/kg and 100 mg/kg. Blood levels of phenylalanine and tyrosine were measured hourly, and the aromatic acid metabolites of phenylalanine were determined in urine. During the chronic in

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Phenylalanine Transport at the Human Blood-Brain Barrierport system in laboratory rats. To test whether phenylalanine transport at the human BBB is also mediated by a very high affinity (low ..) transport system, brain capillaries were isolated from fresh autopsy human brain, as well as from fresh rat brain or from fresh or 42-hour postmortem rabbit brai