Titlebook: Diabetes; An Old Disease, a Ne Shamim I. Ahmad Book 2013 The Editor(s) (if applicable) and The Author(s), under exclusive license to Spring

Ige326

Emerging Type 2 Diabetes in Young Adults,demic driven by the increasing burden of obesity. Evidence is accumulating to suggest that this young diabetic cohort is an aggressive phenotype which leads to the premature development of complications that not only have impact on the quality of life but also unfavourably influence the long term ou

UTTER

indicate

Periodontal Disease and Diabetes,ciency with alteration in the metabolism of lipids, carbohydrates and proteins culminating with different manifestations in different organisms. In humans hyperglycemia is the main consequence of defects in the secretion and/or action of insulin, and its deregulation can produce secondary lesions in

闖入

BARK

Nephropathy in Diabetes, the risk factors of Diabetic Nephropathy (DN). Hyperglycemia-induced metabolic and hemodynamic pathways are recognized to be mediators of kidney injury. Multiple biochemical pathways have been postulated that explain how hyperglycemia causes tissue damage: Non-enzymatic glycation that generates adv

表被動(dòng)

B-cell

accessory

Crohns-disease

Diabetic Autonomic Neuropathy,rom endocrine factors, this neurological disease may cause symptoms, whose differential diagnosis needs a good knowledge of the whole internal medicine. DAN is strongly involved in the development of diabetic foot, ulceration and amputation. The life threatening consequences of cardiac and patient-f

Infinitesimal

,Immunobiology of β-Cell Destruction,of Langerhans. A susceptible genetic background is necessary, but not sufficient, for the development of the disease. Epidemiological and clinical observationsunderscore the importance of environmental factors as triggers of type 1 diabetes, currently under investigation. Islet-specific autoantibodi