Titlebook: Degenerative Retinal Diseases; Matthew M. LaVail,Joe G. Hollyfield,Robert E. Ande Book 1997 Springer Science+Business Media New York 1997

vitreous-humor

https://doi.org/10.1007/978-3-658-06298-9ecognized entity of XLCD associated with the Mizuo-Nakamura phenomenon in which the tapetal-like reflex disappeared after long dark adaptation (3). DNA analysis of XLCD families has also been reported, and the causative gene (COD1) has been localized to the short arm of the X-chromosome (5–7).

衰弱的心

身體萌芽

,TIMP-3 Accumulation in Bruch’s Membrane and Drusen in Eyes From Normal and Age-Related Macular DegeARMD have been described, and end-stage histopathological descriptions of ARMD are available, little is known about the cellular mechanisms associated with the normal maintenance and turnover of Bruch’s membrane, the events that lead to drusen formation, or the cause of subretinal neovascularization and atrophy of the RPE and choriocapillaris.

得體

Photoreceptor Rosettes in Age-Related Macular Degeneration Donor Tissues,ptors. Vision loss occurs gradually over the course of many years. The wet form is less common but results in a more acute visual loss, which involves newly formed blood vessels below the macula that leak fluid and blood into the macular area.

PANT

cacophony

Autoimmune Retinopathy,the cystoid edema present in RP patients that is absent in controls and in patients with RP without cystoid edema. The significance of the antibodies to retinal proteins in the pathophysiology of the disease process is currently under study.

integral

Norrie Disease in Japan,. Some patients have psychomotor retardation and progressive hearing loss as part of a multisystem syndromic disorder. This vitreoretinal dysplastic disease is transmitted as a simple X-linked pattern of inheritance, so that hemizygous males are affected whereas heterozygous females remain asymptomatic..

分離

Theorie der Wirkungen organischer K?rpereen established in a number of studies (1–4), relatively little is known about their composition or origin. Thorough characterization of these age-related deposits has been hampered by a paucity of suitable human donor tissues and the lack of an appropriate animal model of the disease.

Substitution

https://doi.org/10.1007/978-3-8349-4458-0potentials, degree of refractive errors, and family survey (2). It has been concluded that complete and incomplete types are different clinical entities, having demonstrated several explicit differences between the two CSNB types (3,4).

follicular-unit