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Titlebook: Degenerative Retinal Diseases; Matthew M. LaVail,Joe G. Hollyfield,Robert E. Ande Book 1997 Springer Science+Business Media New York 1997

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發(fā)表于 2025-3-21 16:40:06 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Degenerative Retinal Diseases
編輯Matthew M. LaVail,Joe G. Hollyfield,Robert E. Ande
視頻videohttp://file.papertrans.cn/265/264872/264872.mp4
圖書(shū)封面Titlebook: Degenerative Retinal Diseases;  Matthew M. LaVail,Joe G. Hollyfield,Robert E. Ande Book 1997 Springer Science+Business Media New York 1997
描述Since 1984, we have organized Satellite Symposia on Retinal Degenerations that are held in conjunction with the biennial International Congress of Eye Research. The timing and location of our Retinal Degeneration Symposia have all owed scientists and clinicians from around the world to convene and present their exciting new findings. The symposia have been arranged to allow ample time for discussions and one-on-one interactions in a relaxed atmos- phere, where international friendships and collaborations could be established. The II International Symposium (also known as the Sendai Symposium on Retinal Degeneration) was held in 1986 in Sendai, Japan, on the occasion of the retirement of Kat- suyoshi Mizuno as Professor and Chairman of Ophthalmology at Tohoku Medical SchooL On October 5-9, 1996, we returned to Sendai, where the VII International Symposium was held at the Miyagi Zao resort hotel in the beautiful Mt. Zao region of northern Japan. This meeting was held on the occasion of the tenth anniversary of Makoto Tarnai as Pro- fessor and Chairman of Ophthalmology at Tohoku Medical School. One afternoon of the meeting contained a special symposium in honor of Professor Tarnai, wh
出版日期Book 1997
關(guān)鍵詞cell; cell death; cloning; eye; gene therapy; genetics; macular degeneration; retina
版次1
doihttps://doi.org/10.1007/978-1-4615-5933-7
isbn_softcover978-1-4613-7718-4
isbn_ebook978-1-4615-5933-7
copyrightSpringer Science+Business Media New York 1997
The information of publication is updating

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https://doi.org/10.1007/978-1-4615-5933-7cell; cell death; cloning; eye; gene therapy; genetics; macular degeneration; retina
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978-1-4613-7718-4Springer Science+Business Media New York 1997
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Theorie der Wirkungen organischer K?rpersence of drusen and basal laminar deposits (BLD) in the macula. Drusen are extracellular deposits which form between the retinal pigment epithelial (RPE) basal lamina and the inner collagenous zone of Bruch’s membrane. Although the relationship between macular drusen and the development of AMD has b
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https://doi.org/10.1007/978-3-8349-4458-0lectroretinogram (ERG), in which the amplitude of a-wave is normal and larger than that of b-wave (1). We previously reported that CSNB cases with negative ERG can be divided into two types, complete or incomplete, based on the difference in rod visual functions, cone mediated ERGs, ERG oscillatory
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https://doi.org/10.1007/978-3-658-06298-9lor vision disturbance, macular degeneration and abnormalities on cone mediated electroretinogram (ERG) (1–4). This eye disease is distinguishable from the autosomal form by a family pedigree in which affected males appear on the maternal side without male to male transmission. Two XLCD families, wi
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Entscheidungsvorbereitung für den Chefitis pigmentosa (RP) who did not have foveal cysts. Their serum was examined for the presence of antibodies that immunoreacted with retinal proteins. Twenty-seven of 30 patients with bilateral cystoid edema and retinal degeneration had serum anti-retinal protein antibody activity, compared to 3/50 h
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