Titlebook: DNA Methylation: Development, Genetic Disease and Cancer; Walter Doerfler,Petra B?hm Book 2006 The Editor(s) (if applicable) and The Autho

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Epigenotypes of Latent Herpesvirus Genomes,ggesting that histone modifications may control HSV-1 latency in terminally differentiated, quiescent neurons. Epstein-Barr virus (EBV, a human γ-herpesvirus) is associated with a series of neoplasms. Latent, episomal EBV genomes are subject to host cell-dependent epigenetic modifications (DNA methy

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MSL Proteins and the Regulation of Gene Expression,me underlying themes; the molecular components that mediate dosage compensation in both include non-coding RNA molecules, which act as nucleation points for the compensation process. Both systems utilise chromatin-modifying enzymes to remodel large domains of a chromosome. This review will discuss t

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DNA Methylation Profiles of Female Steroid Hormone-Driven Human Malignancies,d not find evidence for global hormone-specific DNA methylation alterations, suggesting that female sex hormones may participate in different tumorigenic pathways that are associated with distinct DNA methylation-based molecular signatures. One such pathway may include . methylation in the context o

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Methylation of Endogenous Human Retroelements in Health and Disease,egions, to separate domains and functional regions within one chromatin domain, to suppress transcriptional noise, and to regulate transcript stability. Retroelements, particularly ALUs, may also fulfill physiological roles during responses to stress and infections. Reactivation and hypomethylation

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Cancer-Linked DNA Hypomethylation and Its Relationship to Hypermethylation, exhibit cancer-associated hypomethylation. However, one of these repeated sequences (NBL2) displayed predominant increases in methylation in some ovarian carcinomas and Wilms tumors and decreases in others. Furthermore, decreases and increases in CpG methylation can be interspersed within a small s