Titlebook: DNA Methylation: Development, Genetic Disease and Cancer; Walter Doerfler,Petra B?hm Book 2006 The Editor(s) (if applicable) and The Autho

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Epigenotypes of Latent Herpesvirus Genomes,NA sequence. Restricted expression of latent, episomal herpesvirus genomes is also due to epigenetic modifications. There is no virus production (lytic viral replication, associated with the expression of all viral genes) in tight latency. . experiments demonstrated that DNA methylation could influe

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MSL Proteins and the Regulation of Gene Expression,tion of gene expression by an epigenetic mechanism. Dosage compensation has evolved to balance the expression of sex-linked genes in males and females, which possess different numbers of sex chromosomes. However, the genetic sequence of the chromosomes is the same in both sexes. This mechanism there

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DNA Methylation Profiles of Female Steroid Hormone-Driven Human Malignancies,sociated with distinct tumorigenic pathways. These pathway-specific DNA methylation changes help define tumor-specific DNA methylation profiles that can be used to further our understanding of tumor development, as well as provide tools for molecular diagnosis and early detection of cancer. Female s

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Genome-wide Analysis of DNA Methylation Changes in Human Malignancies, expression. DNA methylation patterns are heritable and can be passed on to the daughter cell. In this review, we briefly summarize our current knowledge on normal DNA methylation patterns and move on to discuss the current state of the field with respect to altered DNA methylation in cancer. We mak

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Decreased Fidelity in Replicating DNA Methylation Patterns in Cancer Cells Leads to Dense Methylatised fidelity in replicating methylation patters has been analyzed as an underlying mechanism. First we developed a method to analyze the number of errors in replicating CpG methylation patterns in a defined period. A single cell was expanded into 10. cells, and the number of errors during the cultur

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Cancer-Linked DNA Hypomethylation and Its Relationship to Hypermethylation,. Both decreases and increases in DNA methylation are a frequent characteristic of a wide variety of cancers. There is often more hypomethylation than hypermethylation of DNAduring carcinogenesis, leading to a net decrease in the genomic 5-methylcytosine content. Although the exactmethylation change

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