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Titlebook: Current Topics in Heart Failure; Experimental and Cli Rainer W. Gülch,Gerolf Kissling Book 1991 Springer-Verlag Berlin Heidelberg 1991 expe

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樓主: VEER
51#
發(fā)表于 2025-3-30 10:59:11 | 只看該作者
Rentenrechnung und ?quivalenzprinziplead to pathologic hypertrophy. Myocardial fibrosis, the result of cardiac fibroblast growth or abnormal accumulation of fibrillar collagen within the interstitial space, can adversely influence myocardial stiffness and ultimately ventricular function. We have examined the relative importance of ven
52#
發(fā)表于 2025-3-30 15:42:49 | 只看該作者
Funktionen einer unabh?ngigen Variablenent with nifedipine (27 mg/kg b.w./day) and the sympatholytic agent moxonidine (8 mg/kg b.w./day). The treated groups were compared with nontreated SHR and normotensive WKY (n =10 in each group)..When the therapy was started in 6-month old male SHR, blood pressure was increased and left ventricular
53#
發(fā)表于 2025-3-30 19:30:41 | 只看該作者
54#
發(fā)表于 2025-3-30 21:35:06 | 只看該作者
Grenzwerte und Stetigkeit von Funktionenas well as nutritional and hormonal states of the animal Several lines of evidence indicate that the adrenergic nervous system plays an important role in the redistribution of myosin isoforms in the heart. For example, chemical sympathectomy favors the expression of V. isomyosin at the expense of V.
55#
發(fā)表于 2025-3-31 02:36:50 | 只看該作者
https://doi.org/10.1007/978-3-322-93979-1on of muscle represents an important risk factor for cardiovascular disease. However, only little is known of the intracellular consequences of insulin resistance. An important feature of an altered substrate utilization is related to signal transduction of gene expression. For the example of myosin
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