Titlebook: Current Topics in Heart Failure; Experimental and Cli Rainer W. Gülch,Gerolf Kissling Book 1991 Springer-Verlag Berlin Heidelberg 1991 expe

耕種

A new concept for the mechanism of Ca+ +-regulation of muscle contraction. Implications for physiolong) configuration to its strong-binding (force generating) configuration. It is demonstrated that the concept of “rate modulation” requires reinterpretation of force-pCa relations and of the mechanisms of physiological and pharmacological modulation of force-pCa relations. On this basis, an addition

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粗鄙的人

Role of neuroendocrine mechanisms in the pathogenesis of heart failurefailure, atrial natriuretic peptide suppresses the activity of the renin-angiotensin-aldosterone system, which may prevent an increase in peripheral vascular resistance and preserve renal blood flow. In more severe heart failure, the renin-angiotensin-aldosterone system is activated, leading to an i

debunk

Modulation of baroreflex and baroreceptor function in experimental heart failureduced. Three sets of experiments are described. In the first study, the activity from arterial baroreceptors was recorded in normal dogs and in dogs with heart failure. Carotid sinus pressure-receptor discharge curves were constructed along with pressure-diameter curves. Increasing carotid sinus pre

Blood-Vessels

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https://doi.org/10.1007/978-3-662-30769-4experiment; heart; heart failure

Dungeon

978-3-7985-0894-1Springer-Verlag Berlin Heidelberg 1991

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尖牙

https://doi.org/10.1007/978-3-322-91961-8nabling the whole organ to cope with increased load, as a rule also include unfavorable components. This is primarily due to competition between different demands with respect to, for instance, mechanical or energetic parameters. An impressive demonstration of the ambivalence of adaptive processes c

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