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Titlebook: Color Atlas of Human Fetal and Neonatal Histology; Linda M. Ernst,Eduardo D. Ruchelli,Dale S. Huff Book 2019Latest edition Springer Nature

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31#
發(fā)表于 2025-3-27 00:09:56 | 只看該作者
https://doi.org/10.1007/978-3-322-94072-8ntestinal tract is established in embryonic life, the layers of the wall continue to develop during fetal life. Thus, the histology of the gastrointestinal tract is characterized by numerous changes in the fetal period, which are highlighted in this chapter.
32#
發(fā)表于 2025-3-27 04:33:44 | 只看該作者
33#
發(fā)表于 2025-3-27 08:57:38 | 只看該作者
Heartse differences is important when examining the fetal heart. This chapter highlights the histological features of the fetal and neonatal myocardium as well as the specialized structures such as cardiac valves and the cardiac conduction system.
34#
發(fā)表于 2025-3-27 09:55:19 | 只看該作者
35#
發(fā)表于 2025-3-27 13:44:29 | 只看該作者
36#
發(fā)表于 2025-3-27 19:10:17 | 只看該作者
Liverimesters of gestation. This chapter reviews the histological changes in the fetal liver evident in hepatocytes, the sinusoidal lining cells, the intrahepatic biliary tree, and the hematopoietic elements and discusses the development of the hepatic vasculature and extrahepatic biliary tree.
37#
發(fā)表于 2025-3-27 22:09:12 | 只看該作者
Pancreasocrine, and endocrine components undergo throughout gestation. The unique features of the endocrine tissue of the fetal and neonatal pancreas must be considered before drawing conclusions as to their possible causative role in either presumed or clinically established hyperinsulinism.
38#
發(fā)表于 2025-3-28 05:59:24 | 只看該作者
39#
發(fā)表于 2025-3-28 09:05:59 | 只看該作者
Kidneyt until the end of gestation, when the formation of nephrons (nephrogenesis) comes to an end. A lack of familiarity with these differences may lead to mistakenly considering normal findings as pathologic ones, or to overlooking abnormalities of renal development, such as impaired nephrogenesis with
40#
發(fā)表于 2025-3-28 12:56:58 | 只看該作者
Urinary Bladderase, or renal tubular dysplasia) will result in hypoplasia of the bladder. Conversely, the fetal bladder becomes enlarged and hypertrophic in the presence of prolonged obstruction of the bladder outlet or urethra. These pathologic changes should be distinguished from the normal variability of physio
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