Titlebook: Color Atlas of Human Fetal and Neonatal Histology; Linda M. Ernst,Eduardo D. Ruchelli,Dale S. Huff Book 2019Latest edition Springer Nature

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The Scandinavian Trade with China,t until the end of gestation, when the formation of nephrons (nephrogenesis) comes to an end. A lack of familiarity with these differences may lead to mistakenly considering normal findings as pathologic ones, or to overlooking abnormalities of renal development, such as impaired nephrogenesis with

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https://doi.org/10.1007/978-981-13-0332-6ase, or renal tubular dysplasia) will result in hypoplasia of the bladder. Conversely, the fetal bladder becomes enlarged and hypertrophic in the presence of prolonged obstruction of the bladder outlet or urethra. These pathologic changes should be distinguished from the normal variability of physio

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https://doi.org/10.1007/978-981-13-0332-6y periods of relative inactivity. Each step is triggered by a rapid rise in production of gonadotropins and androgens, which triggers a new step in maturation of germ cells, Leydig cells, and Sertoli cells. The first step begins at the end of the 6th week postfertilization; the second, during the 14

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Daniel Adams,Terry L. Alford,James W. Mayerd is the largest of these three glands. The prostate gland has both epithelial and stromal components that can be recognized histologically, and the early development of these components is seen during fetal life. Aberrations of the proper development of the prostate gland can be seen in some syndro

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