Titlebook: Clinical Relevance of Genetic Factors in Pulmonary Diseases; Takeshi Kaneko Book 2018 Springer Nature Singapore Pte Ltd. 2018 Smoking.Bron

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Breeding Efforts and Biotechnologyve revealed the genetic components of common diseases. Genetic mapping is an unbiased method to comprehensively identify genes and biological pathways involved in diseases or traits. Genome-wide association studies (GWASs) have convincingly identified disease-associated loci. Most of the associated

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https://doi.org/10.1007/978-3-319-75671-4thma and for identifying targets of treatment for the syndrome. As with studies of other common complex diseases, genetic studies of asthma have led to considerable advances in the understanding of this disease. Genome-wide association studies have greatly advanced the identification of the most imp

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Amrita Srivastav,Tushar Khare,Vinay Kumar This is because approximately 15% of the smoking population eventually suffer from this disease, while the others sustain normal lungs despite their smoking habit. A well-known causal gene of COPD is the serine protease inhibitor A1 (SERPINA1) gene, which causes hereditary severe α1-antitrypsin (A1

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Seville Flowers,Frederick R. Evans. HHT is often found to be the underlying cause of pulmonary arteriovenous malformations (PAVMs). Mutations in ., ., ., ., ., and . have been identified in cases of HPAH. Mutations in ., ., and . have been identified in cases of HHT. The average penetrance of . mutations is only 30% in HPAH. In cont

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Irrigation with Saline Water in Pugliaoped countries and now being more prevalent than tuberculosis. Severe mycobacterial disease is mostly confined to patients who are immunocompromised either by acquired or inherited causes. Genetic aberrations in pathways critical for host defense against mycobacteria—which involve functional interle

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Bacterial Reduction of Selenium, germline mutations, inherited gene alterations, and single-nucleotide polymorphisms. Germline mutations and inherited gene alterations are related to the development of familial lung cancer, and certain single-nucleotide polymorphisms are associated with an increase in the risk of lung cancer. Othe