Titlebook: Clinical Nephrotoxins; Renal Injury from Dr Marc E. Broe,George A. Porter,Gilbert Deray Book 2008Latest edition Springer-Verlag US 2008 Int

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Animal models for the assessment of acute renal dysfunction and injury ated when interpreting the results. However certain models are useful for studying specific types of neprotoxic injury. For example, techniques for study of the microcirculation are ideal to study drugs that cause acute renal dysfunction on a vascular basis. These drugs include prostaglandin inhibi

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Renal cell culture models: , and the presence of xenobiotic metabolizing enzymes such as cytochrome P-450, NADPH-cytochrome c reductase, glucuronyl transferase, sulfotransferases, glutathione S-transferases, cysteine conjugate β-lyase, monooxygenases and prostaglandin H synthase [3]. Another factor is the intracellular concentr

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Aminoglycosides and vancomycin bactericidal efficacy increases with increasing aminoglycoside concentration. Aminoglycosides also exhibit a post-antibiotic effect meaning they continue to kill bacteria even after the aminoglycoside concentration has fallen below the bacterial minimum inhibitory concentration. Another useful attr

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Book 2008Latest editione nephrologist, internist, general practitioner, toxicologist, pharmacologist, anesthesiologist, epidemiologist, public health official, pharmaceutical industry, and national drug safety committees, among others..

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