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Titlebook: Clinical Nephrotoxins; Renal Injury from Dr Marc E. Broe,George A. Porter,Gilbert Deray Book 2008Latest edition Springer-Verlag US 2008 Int

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發(fā)表于 2025-3-21 19:43:31 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Clinical Nephrotoxins
副標題Renal Injury from Dr
編輯Marc E. Broe,George A. Porter,Gilbert Deray
視頻videohttp://file.papertrans.cn/229/228090/228090.mp4
概述Premier reference on nephrotoxins.Includes supplementary material:
圖書封面Titlebook: Clinical Nephrotoxins; Renal Injury from Dr Marc E. Broe,George A. Porter,Gilbert Deray Book 2008Latest edition Springer-Verlag US 2008 Int
描述.Clinical Nephrotoxins: Renal Injury from Drugs and Chemicals, Third Edition is a comprehensive text on all aspects of adverse effects by drugs, chemical substances and radiation on the kidneys. The importance of the toxicity of drugs and other substances for the kidneys is increasingly recognized. The book consists of themes such as clinical relevance, renal handling and cellular mechanisms of nephrotoxicity, as well as animal and cell culture models. In addition, the volume highlights specific types of drugs, such as anti-infectious agents and anti-inflammatory, cardiovascular and anti-cancer drugs. The last section deals with prevention and focuses on urinary biomarkers, pharmacological aspects and drug dosage in renal failure. In this third and completely revised edition, several new topics were added and additional figures and tables will be included...The book is of interest for the nephrologist, internist, general practitioner, toxicologist, pharmacologist, anesthesiologist, epidemiologist, public health official, pharmaceutical industry, and national drug safety committees, among others..
出版日期Book 2008Latest edition
關(guān)鍵詞Internist; antibiotic; intensive care; interferon; kidney; nephrotoxins, renal injury, drugs, urinary bio
版次3
doihttps://doi.org/10.1007/978-0-387-84843-3
isbn_softcover978-1-4899-7728-1
isbn_ebook978-0-387-84843-3
copyrightSpringer-Verlag US 2008
The information of publication is updating

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Psychic Activity and the Brain,from 29,269 critically ill patients in the ICU from 54 study centers that 30% of patients had renal dysfunction upon entering in the ICU and the prevalence of AKI defined by the need for dialysis to be 6% [3]. Mehta reporting on the PICARD experience (Program to Improve Care in Acute Renal Disease)
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S. Le?niewski’s Lecture Notes in Logicsive as possible. Any suspicions of an expected or a new problem with a medicine should be well reported, signalled and evaluated. Despite extensive testing of medicines before they are approved for marketing, unexpected and/or rare adverse drug reactions may occur when the medicine is used in norma
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https://doi.org/10.1007/978-3-319-71743-2dicators of specific anatomic, physiologic, biochemical, or molecular events; [2] that thay are associated with normal biological processes or accompany the onset, progression and/or severity of specific pathological or toxic conditions and [3] are that they are useful for measuring the progress of
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https://doi.org/10.1007/978-3-319-71743-2ogy of renal diseases. This points out new clues on the mechanisms by which chemically-induced immune response trigger immune nephropathies. We will describe the T-cell subsets including Th1 and Th2 cells that may be implicated in renal inflammation. The role of Th1 and Th2 CD4. T-cell subsets in th
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