Titlebook: Clinical Diagnosis of Atherosclerosis; Quantitative Methods M. Gene Bond (Associate Professor of Comparative M Textbook 1983 Springer-Verla

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書目名稱Clinical Diagnosis of Atherosclerosis影響因子(影響力)




書目名稱Clinical Diagnosis of Atherosclerosis影響因子(影響力)學(xué)科排名




書目名稱Clinical Diagnosis of Atherosclerosis網(wǎng)絡(luò)公開度




書目名稱Clinical Diagnosis of Atherosclerosis網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Clinical Diagnosis of Atherosclerosis被引頻次




書目名稱Clinical Diagnosis of Atherosclerosis被引頻次學(xué)科排名




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書目名稱Clinical Diagnosis of Atherosclerosis年度引用學(xué)科排名




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書目名稱Clinical Diagnosis of Atherosclerosis讀者反饋學(xué)科排名

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https://doi.org/10.1057/9781403907172toms that simulate arteries in the millimeter range (1). While these noninvasive methods appear very likely to be used for quantitative lesion assessment in the near future, relatively little quantitative experience has been gained to date. Conversely, computer methods to quantify atherosclerosis fr

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https://doi.org/10.1057/9780230372931that lesion has increased to approximately 20% of the total mass. However, in carefully dissected, large, raised lesions described as atherosclerotic plaques, the percent dry weight is often greater than 50% (2). Since the density of the lipids is some 30–40% less than the density of the other const

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https://doi.org/10.1007/978-1-349-05265-3ection. Severe, uncompensated obstructions, on the other hand, compromise the viability of the tissues even at rest or in the absence of extraneous trauma--leading to ischemic rest pain, nonhealing ulcers, tissue destruction by unchecked infection, and eventual gangrene. Although lesions capable of

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https://doi.org/10.1007/978-3-319-70341-1 components during lesion initiation, progression, and regression. An appreciation of the biochemical and subcellular changes and spatial relations of lesion components at various points in time is essential to understanding the pathogenesis of atherosclerosis. The importance of these relationships

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Quantitating Atherosclerosis self-evident that controversies regarding the relative accuracy, sensitivity and specificity of each of the detection methods can ultimately be resolved only by quantitative comparisons of the images with the actual, corresponding lesions. Secondly, we have entered an era in which we wish to follow

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Atherosclerosis Quantitation by Computer Image Analysistoms that simulate arteries in the millimeter range (1). While these noninvasive methods appear very likely to be used for quantitative lesion assessment in the near future, relatively little quantitative experience has been gained to date. Conversely, computer methods to quantify atherosclerosis fr