Titlebook: Clinical Diagnosis of Atherosclerosis; Quantitative Methods M. Gene Bond (Associate Professor of Comparative M Textbook 1983 Springer-Verla

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Physical Biochemistry of the Lesions of Man, Subhuman Primates, and Rabbitsthat lesion has increased to approximately 20% of the total mass. However, in carefully dissected, large, raised lesions described as atherosclerotic plaques, the percent dry weight is often greater than 50% (2). Since the density of the lipids is some 30–40% less than the density of the other const

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Correlation of Lesion Configuration with Functional Significanceection. Severe, uncompensated obstructions, on the other hand, compromise the viability of the tissues even at rest or in the absence of extraneous trauma--leading to ischemic rest pain, nonhealing ulcers, tissue destruction by unchecked infection, and eventual gangrene. Although lesions capable of

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Correlation of Postmortem Angiography with Pathologic Anatomynd have come to the conclusion that angiography in vivo underestimates stenoses (2–6). This discrepancy is thought to occur because plaques cause arterial lumens to be “eccentric and slit-like” (2,6) rather than rounded, so that many angiographic projections may show deceptively wide lumens. In most

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nfirmed in vivo by ultrasonic and other imaging techniques. The morbid anatomist is challenged anew to describe lesions as they are likely to occur in vivo. To achieve closer correlation with natural conditions, perfu- sion fixation of arteries under arterial pressure is becoming more widely used and has alre978-1-4684-6279-1978-1-4684-6277-7

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https://doi.org/10.1057/9781403907172ectional histological slides. These methods are adequate for testing hypotheses that are aimed at determining large differences in the amount of atherosclerosis either between or among groups or within individual groups of subjects. However, these methods are considerably less sensitive in describing smaller or more subtle differences.

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https://doi.org/10.1007/978-3-319-70341-1etection and quantification of atherosclerotic lesions in human and nonhuman primate carotid and iliofemoral arteries. In 1981 this resulted in the award of seven contracts to establish one animal center, five clinical centers and a coordination center (Table 1).

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