Titlebook: Chemical Carcinogenesis; Models and Mechanism Francesco Feo,Paolo Pani,Renato Garcea Book 1988 Springer Science+Business Media New York 198

Osmosis

Mast-Cell

發(fā)芽

https://doi.org/10.1007/978-1-4757-9640-7apoptosis; cancer; carcinogenesis; cell; cell culture; etiology; hormones; liver; liver tumor; mutagen; pathog

賄賂

Jocose

黃瓜

Interaction of Activated N-Nitrosamines with DNAise at sites in DNA of unusually high reactivity for ultimate carcinogens, or whether carcinogen interaction is random. This information is not available for simple methylating agents such as those derived from N-nitroso compounds.

Ambulatory

http://image.papertrans.cn/c/image/224295.jpg

轉(zhuǎn)換

Cytochromes P-450 as Determinants of Susceptibility to Carcinogenesis by Aromatic Amines and Nitroarytochromes P-450 have been shown to play a major role in this process (reviewed in ref. 8). In this paper, the catalytic activity of specific cytochrome P-450 isozymes for the activation of these carcinogens will be discussed in relation to species and individual differences in cancer susceptibility

山羊

跳脫衣舞的人

Relationship Between Structure and Mutagenic/Carcinogenic Activity of Short Chain Aliphatic Halocomptivity of halocompounds decreases from iodo- to bromo- and chloro-substituted compounds. More generally, halocompounds are enzymatically “activated” to interact with macromolecules. The first metabolic step is an oxidation due to the mixed function oxidase system (MFO) in the presence of cytochrome