Titlebook: Central Cholinergic Synaptic Transmission; Michael Frotscher,Ulrich Misgeld Book 1989 Birkh?user Verlag 1989 Nervous System.Parkinson.anat

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Cholinergic mechanisms in the telencephalon of cat and chickenet al., 1987; Rigdon and Pirch, 1986), as well as their role in attentional processing (Phillis, 1968; Singer, 1979). These afferents originate predominantly from several nuclei in the basal forebrain (Mesulam et al., 1983). Similar cholinergic afferents are known from non-mammalian species, e.g. fr

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M. Meister,C. Hetru,J. A. Hoffmannonists. This finding suggests that stimulation of PI turnover may be involved in the blockade of the M-current. Although activation of protein kinase C with phorbol esters has little effect on the M-current, intracellular application of inositol trisphosphate did reduce the M-current. We were unable

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J. P. Rast,Z. Pancer,E. H. Davidsont a single, primary muscarinic action, the acceleration of phosphinositide turnover, may account for a variety of secondary effects: on the one hand, via activation of PKC, a number of possible PKC-mediated actions, such as block of the slow AHP; on the other, via IP. formation, a block of I. and a

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https://doi.org/10.1007/978-3-642-59717-6d as an inward current resulting from a decrease in conductance which was constant over the membrane potential range of -100 to -50 mV. Reversal potential was negative to -80 mV. An increase in the excitability of neurons (as measured by responses to identical intracellularly applied depolarizing cu