Titlebook: Cellular Primary Immunodeficiencies; Mario Milco D‘Elios,Cosima Tatiana Baldari,Frances Book 2021 Springer Nature Switzerland AG 2021 T Ce

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書目名稱Cellular Primary Immunodeficiencies影響因子(影響力)




書目名稱Cellular Primary Immunodeficiencies影響因子(影響力)學(xué)科排名




書目名稱Cellular Primary Immunodeficiencies網(wǎng)絡(luò)公開度




書目名稱Cellular Primary Immunodeficiencies網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cellular Primary Immunodeficiencies被引頻次




書目名稱Cellular Primary Immunodeficiencies被引頻次學(xué)科排名




書目名稱Cellular Primary Immunodeficiencies年度引用




書目名稱Cellular Primary Immunodeficiencies年度引用學(xué)科排名




書目名稱Cellular Primary Immunodeficiencies讀者反饋




書目名稱Cellular Primary Immunodeficiencies讀者反饋學(xué)科排名

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Primary Immunodeficiencies,Primary immunodeficiency diseases (PID) are a part of the growing field of inborn errors of immunity (IEI), sharing a raised susceptibility to infectious diseases and often associated to other features such as autoimmunity, autoinflammation, and lymphoproliferation and susceptibility to neoplastic disease and atopic diseases.

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Mario Milco D‘Elios,Cosima Tatiana Baldari,FrancesFeatures updated management and all the latest genetic sof cellular primary immunodeficiencies.Offers in-depth description of the basis of cellular immune responses.Valued companion to immunologists,

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https://doi.org/10.1007/978-3-540-49761-5pecific MHC-associated antigen, T lymphocytes assemble a highly organized intercellular junction, referred to as the immune synapse, on which T cell activation, proliferation, and differentiation crucially depend. Immune synapse assembly and function are impaired in a subgroup of human immunodeficie

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https://doi.org/10.1007/978-3-540-49761-5anical and chemical means, they prevent invasion by pathogens. The mucosal immune system consists of an innate and acquired system that interact with each other in a complex way. The mucosal immune system also has the delicate task of differentiating between pathogens and non-pathogens. The T and B

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Equipment, Instruments, and Materials,nogenic defects affecting phagocyte numbers and/or function have been described. A common hallmark of all congenital defects of phagocytes is an increased susceptibility to severe bacterial and fungal infections. Typical sites of infection are the skin, the oral mucosa and gingiva, lymph nodes, the