| 書(shū)目名稱(chēng) | Cardiac Left Ventricular Hypertrophy | | 編輯 | H. E. D. J. Keurs,J. J. Schipperheyn | | 視頻video | http://file.papertrans.cn/222/221785/221785.mp4 | | 叢書(shū)名稱(chēng) | Developments in Cardiovascular Medicine | | 圖書(shū)封面 |  | | 描述 | Left ventricular hypertrophy (LVH) is usually considered to be a compen- satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is damaged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden deat | | 出版日期 | Book 1983 | | 關(guān)鍵詞 | arrhythmia; artery; circulation; congenital heart disease; echocardiography; heart; heart disease; hyperten | | 版次 | 1 | | doi | https://doi.org/10.1007/978-94-009-6759-5 | | isbn_softcover | 978-94-009-6761-8 | | isbn_ebook | 978-94-009-6759-5Series ISSN 0166-9842 | | issn_series | 0166-9842 | | copyright | Martinus Nijhoff Publishers, Boston 1983 |
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