Titlebook: Cardiac Left Ventricular Hypertrophy; H. E. D. J. Keurs,J. J. Schipperheyn Book 1983 Martinus Nijhoff Publishers, Boston 1983 arrhythmia.a

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0166-9842 es of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden deat978-94-009-6761-8978-94-009-6759-5Series ISSN 0166-9842

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Book 1983rsible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden deat

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Epidemiologic Features of Left Ventricular Hypertrophy in Normotensive and Hypertensive Subjects early 1950’s was about 2%; it declined to less than 1% among the offspring subjects when they were about the same age as the original cohort in the early 1950’s..In the 1979–1982 study, echo-LVH was found in 6% of the offspring study subjects and in 22% of the older original cohort subjects. The pr

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Determinants and Modulators of Left Ventricular Structureesis..Volume overload in pregnancy, anemia, renal failure and, most commonly, obesity is an additional hypertrophogenic factor increasing wall stress by increasing LV chamber volume. The combination of hypertension and obesity is particularly unfavorable for the heart..Aging in itself appears to be

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Relationship Between Cardiac Work and Cardiac Growth: Some General Thoughts on Cardiac Hypertrophylation of glycogen Phosphorylase b and other Ca.-stimulated processes..One consequence of this conclusion, directly applicable to hypertrophy, is that calcium may control biochemical mechanisms for protein synthesis presumably at the level of DNA-transcription. An important precedent was recently se

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Myocardial cell Properties and Hypertrophyred with a strain gauge. Muscle length was measured and controlled with a servomotor. Sarcomere length was measured and controlled by means of laser diffraction techniques. Mechanical recovery curves in controls and hearts following hypobaric hypoxia showed a rapid rise in the first 600 ms followed

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