Titlebook: Beh?et‘s Disease; From Genetics to The Yoshiaki Ishigatsubo Book 2015 Springer Japan 2015 Autoimmune.Uveitis.anti-TNF-alpha antibodies

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Neurological Involvement,w-dose methotrexate (MTX) has been shown to decrease cerebrospinal fluid IL-6 levels without progression of neuropsychological manifestations and is now a gold-standard treatment. Recent studies have demonstrated that infliximab has a beneficial effect in chronic progressive NB patients with inadequ

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Mucocutaneous Manifestations,class I chain-related gene A (MICA) by stress in active BD patients as the intrinsic factors. . gene and 65kD of heat shock protein (HSP-65) derived from oral . (. are supposed to play an important role as the extrinsic factor in BD pathogenesis. The peptides of . gene are highly homologous with the

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urce for both basic researchers and clinical physicians?.This book, besides reviewing basic and clinical aspects of Beh?et‘s disease, covers the latest findings, including genetic studies and treatment with biologics for the disease. .Although the cause of Beh?et‘s disease is still unknown, it is we

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Book 2015 biologics for the disease. .Although the cause of Beh?et‘s disease is still unknown, it is well known that genetic factors, such as HLA-B51, are involved in its development. Recently, novel susceptibility loci including IL10, IL23R-IL12RB2, and endoplasmic reticulum aminopeptidase 1 have been ident

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An Introduction to Morningness-Eveningness,cacy and limitation of anti-TNF therapy for BD patients..Lastly, we would like to show our trial to establish an animal model with disease susceptible genes which have been identified by genome-wide association study and subsequent investigation, leading to a novel therapeutic strategy for more appropriate targets in Beh?et’s disease.

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,The Immunopathology of Beh?et’s Disease,differentiation, antigen-presenting cell (APC) activation, and their contribution to the pathogenesis of BD, especially in view of IL12/IL23 family cytokine production and pattern recognition receptor (PRR) function of macrophages/monocytes.

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Perspective,cacy and limitation of anti-TNF therapy for BD patients..Lastly, we would like to show our trial to establish an animal model with disease susceptible genes which have been identified by genome-wide association study and subsequent investigation, leading to a novel therapeutic strategy for more appropriate targets in Beh?et’s disease.

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An Introduction to Morningness-Eveningness,sed to manage ocular inflammatory attacks. Recently, antitumor necrosis factor-α agents have been shown to dramatically reduce the frequency of ocular attacks in BD patients with refractory uveoretinitis, indicating it as a promising therapy for ocular BD.