Titlebook: Beh?et‘s Disease; From Genetics to The Yoshiaki Ishigatsubo Book 2015 Springer Japan 2015 Autoimmune.Uveitis.anti-TNF-alpha antibodies

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An Introduction to Morningness-Eveningness,um. Although mucocutaneous manifestations are self-limiting, ocular involvement can cause blindness. Involvement of large vessels, gastrointestinal tract, and central nervous system is less frequent but can be life-threatening and leave irreversible damage..Because of heterogeneity of clinical manif

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Morningness-Eveningness in Adolescence,h a particular genetic background. It is well established that BD is strongly associated with the human leukocyte antigen (HLA) class I allele, ., in many different ethnic groups, indicating that the . allele is one of the genetic factors underlying BD. However, the presence of .-negative BD patient

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Morningness-Eveningness in Adolescence,gnostic criteria for both types of NB have been recently established based on the results of a multicenter retrospective survey on BD patients with neurological manifestations in Japan. Administration of corticosteroids is usually necessary and effective in attacks of acute NB, although they are som

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An Introduction to Morningness-Eveningness,tem organs. We here reviewed the mucocutaneous manifestations including recurrent aphthous stomatitis (RAS) and the several important advances in our understanding of BD pathogenesis based on the genetically intrinsic and extrinsic triggering factors. Most of BD patients tend to have hypersensitivit

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An Introduction to Morningness-Eveningness,s successfully treated with emergent endovascular intervention followed by infliximab. Case 2 repeated acute neurological attacks during infliximab therapy for preexisting ocular involvement. Sustained elevation of IL-6 in cerebrospinal fluid suggested transition to chronic progressive type of neuro