Titlebook: Autoimmune Diseases in Endocrinology; Anthony P. Weetman Book 2008 Humana Press 2008 Diabetes.Diabetes mellitus.Endocrinology.Internal med

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https://doi.org/10.1007/978-3-658-33751-3ancreas by autoreactive CD4. and CD8. T lymphocytes. Animal models of the spontaneous disease as well as genetically modified models have contributed significantly to our understanding of both the pathogenesis and the pathophysiology of the disease. In particular, animal models have allowed a better

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Gestaltung der Erfolgsrechnung nach IFRShave other organ-specific autoimmune diseases such as thyroiditis or celiac disease (CD), most T1D patients suffer from life-long insulin dependence because only the β cells have been eradicated. The genetic etiology is strongly associated with certain HLA-DQ class II heterodimers, which in part may

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,Führungshandlungen im Krankenhaus,lateral involvement of the adrenal cortex, together with a deficiency in the production of glucocorticoids, mineralocorticoids, and androgens, associated with high levels of both adrenocorticotropin hormone and plasma renin activity. The prevalence of the disease is 110–140 cases per million, and au

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Basic Mechanisms in Autoimmunity difficult to prove in the complex human system. The most intriguing hypotheses and the mechanisms that control T-cell activation and regulation, which have been studied in vitro and in experimental animal models, will be discussed in this chapter together with their relevance to human diseases.

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Contemporary Endocrinologyhttp://image.papertrans.cn/b/image/166144.jpg

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Autoimmune Diseases in Endocrinology978-1-59745-517-6Series ISSN 2523-3785 Series E-ISSN 2523-3793

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,Führungshandlungen im Krankenhaus, disease and how we can predict it with a degree of certainty and how we are now trying to modulate the disease process. Lessons learned from autoimmune diabetes could be relevant to other autoimmune diseases.