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Titlebook: Autoimmune Diseases in Endocrinology; Anthony P. Weetman Book 2008 Humana Press 2008 Diabetes.Diabetes mellitus.Endocrinology.Internal med

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發(fā)表于 2025-3-21 19:39:05 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Autoimmune Diseases in Endocrinology
影響因子2023Anthony P. Weetman
視頻videohttp://file.papertrans.cn/167/166144/166144.mp4
發(fā)行地址Conveniently divided into four, easy-to-follow sections.Novel text on key features that predispose individuals to autoimmune diseases.In-depth discussion of basic epidemiology, genetic and environment
學(xué)科分類Contemporary Endocrinology
圖書封面Titlebook: Autoimmune Diseases in Endocrinology;  Anthony P. Weetman Book 2008 Humana Press 2008 Diabetes.Diabetes mellitus.Endocrinology.Internal med
影響因子.Autoimmune Diseases in Endocrinology. is a comprehensive and novel text that examines key features that predispose individuals to autoimmune diseases. The text begins with three introductory chapters which provide sufficient information so that anyone, including those without a background in recent immunology, will be able to understand the developments in the field. The next and largest section of the book concerns autoimmune thyroid disease, and authors of these chapters take the reader through the basic epidemiology, genetic and environmental risk factors, immunopathogenesis, and the diagnostic and management aspect of the disease. The next section discusses Type 1 diabetes mellitus. In the final section, authors explain other autoimmune endocrinopathies, including Addison’s disease, premature ovarian failure, autoimmune hypophysitis, and autoimmune polyglandular syndrome types 1 and 2. Comprehensive and timely, .Autoimmune Diseases in Endocrinology. will become an essential reference for all endocrinologists and internal medicine practitioners working in this field..
Pindex Book 2008
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Gestaltung agiler Arbeitsweltenoimmune targets are thyroglobulin (TG), thyroid peroxidase (TPO) and the thyrotropin receptor (TSHR). Autoantibodies and specific T cells directed against all three autoantigens can be detected in the circulation of HT and GD patients and also in a significant proportion of the healthy population. I
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,Fallstudie – Lufthansa New Workspace,mune disease. Fifty years after the initial description of thyroid autoimmunity and identification of the major autoantigens, the disease remains the best understood form of human organ-specific autoimmunity. The structure and function of the major thyroid antigens, together with recent advances in
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https://doi.org/10.1007/978-3-658-16311-2 There is a genetic predisposition to Graves’ disease, determined by alleles at the major histocompatibility complex (.), cytotoxic T-lymphocyte-associated antigen (.), protein tyrosine phosphatase non-receptor 22 (.), and other less well-defined chromosomal loci. Additional, non-genetic, factors th
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https://doi.org/10.1007/978-3-658-16311-2nsufficient thyroid function caused by an autoimmune thyroid diseases autoimmune destruction of the thyroid gland. The most common form of autoimmune thyroid diseases is chronic or lymphocytic autoimmune thyroiditis (Hashimoto’s thyroiditis). Prevalence of autoimmune hypothyroidism is high. In its i
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Flexible disponierte Arbeitszeitsysteme,s of Graves’ disease, which should be viewed as a multisystem autoimmune disease involving thyrocytes but also orbital and pretibial fibroblasts. Smoking is a risk factor for TAO, and cessation of smoking is useful in the primary, secondary and tertiary prevention of TAO. The immunopathogenesis of T
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