Titlebook: Apoptosis Techniques and Protocols; Andréa C. LeBlanc Book 2002Latest edition Humana Press 2002

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,Apoptotic and Oxidative Indicators in Alzheimer’s Disease,far more complex picture, in which apoptotic and necrotic phenotypes might present simultaneously (.). For example, TUNEL positivity in cardiomyocytes does not necessarily entail the presence of cell death that has an apoptotic phenotype (.). Complicating the matter further is the promiscuous use of

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https://doi.org/10.1007/978-3-642-91515-4isms of cell death, through apoptosis, have been elucidated and increasingly point toward mitochondria as the gatekeepers of apoptosis. The morphological changes that define apoptosis can be attributed to the actions of a family of cysteinyl-dependent aspartate specific proteinases, called caspases

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https://doi.org/10.1007/978-3-642-91515-4ke the proper connections with their postsynaptic targets die naturally during development due to the lack of sufficient trophic support (.). Cell demise is operated in a wellordered fashion and culminates in the activation of a network of specific proteases, the caspases that execute the death prog

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Elemente der Sechsten Hauptgruppeole in glycolysis. Indeed, the most frequent reference to GAPDH in recent scientific literature is as the “housekeeping” gene used to standardize Northern blots. A careful examination of the literature of the last 25 years, however, reveals a number of novel actions of GAPDH beyond a role in glycoly

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H. Garschagen,W. Kimpel,J. Weisenaling and axonal transport. Previous chapters have focused on the preparation of compartmented cultures. Here we focus on analytical approaches that we have used with compartmented cultures that can be used to investigate apoptosis, intracellular signaling, and other issues. Techniques used for inc

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